Fig. 6. Genetic interactions with other rotational genes. (A-F) Disrupting Egfr signalling has no effect on the nemo^P1 phenotype. Top panels show sections through adult eyes; bottom panels are schematics of these images. (A,B) nemo^P1. All ommatidia are arrested at approximately 45°. (C,D) sev-Gal4, UAS-keren/+; nemo^P1. (E,F) ru¹, nemo^P1. Conditions of both overactive (C) and underactive (E) Egfr signalling fail to modify the nemo^P1 phenotype. (G,H) sca¹ mutants show relatively minor defects in ommatidial rotation in the adult eye. Most correctly specified ommatidia are orientated at 90° to the equator, with only a few being misrotated. Colour coding in schematics is as previously (black, correctly orientated; green, underrotated; blue, overrotated; black circles, mis-specified ommatidia; red line, equator).