Fig. 8. hbs antagonizes sns function during myoblast fusion and gut morphogenesis. Wild type (A-C) or embryos with genotypes hbs²⁵⁹³/Df(2R)X28 (D-F), hbs⁴⁵⁹/Df(2R)X28 (G-I) and sns^XB3 hbs²⁵⁹³/hbs⁴⁵⁹ (J-L) are shown. Heart (black arrowheads, A,D,G,J) and gut (B,E,H,K) views of stage 16 embryos stained for Myosin, and lateral views of stage 12 embryos stained for Fasciclin III (C,F,I,L). hbs homozygous mutant embryos showed not only a partial fusion block, particularly conspicuous around the heart (compare A with D,G white arrowheads), but also an aberrant morphology of the gut chambers (asterisks) in late embryos (compare B with E,H), and gaps in the visceral mesoderm (black arrowheads; compare C with F,I). Both aspects were suppressed by halving the sns genetic dose (compare D-F with J-L).

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