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Fig. 2. nmo antagonizes Wg signaling during wing development. (A) A wild-type adult wing. (B) sd-Gal4/UAS-nmo. (C) zw3^m11-1/+; sd-Gal4/UAS-nmo. (D) The null allele nmo^DB24. (E) sd-Gal4/UAS-DFz2N. (F) Loss of nmo in sd-Gal4/UAS-Fz2N; nmo^DB24/nmo^DB24 flies rescues the severe wing defect seen in E. (G) sd-Gal4/+; UAS-Daxin causes a wing-to-notum transformation (see inset). (H,I) Reductions in nmo rescue in a dose-dependent manner in (H) sd-Gal4/+; UAS-Daxin, nmo^DB24/+ and (I) sd-Gal4/+; UAS-Daxin, nmo^DB24/nmo^DB24. (J) ap-Gal4/+; UAS-Daxin. Reductions in nmo rescue this phenotype in a dose-dependent manner. (K) ap-Gal4/+; UAS-Daxin, nmo^DB24/+ and (L) ap-Gal4/+; UAS-Daxin, nmo^DB24/nmo^DB24. (M) 71B>flu ${Delta}$ arm. (N) 71B>nmo. (O) UAS-flu ${Delta}$ arm/UAS-nmo; 71B-Gal4/+.

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